By Jiang Guomin, Yu Xiaolin and Cai Rongchang

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Additional info for The Handbook of Insecticide Formulations and Its Technologies for Household and Public Health Uses

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11). Anti-A can be readily adsorbed onto and eluted from Ax cells. 003% of group A) [275] and as 1 in 40 000 [281]. GTA cannot usually be detected in Ax serum or red cell membranes [212,279–281]. H-transferase activity in Ax sera is low [281]. The molecular genetics of Ax reflects the heterogeneity of the serological phenotypes. The most common Ax allele (Ax01) has the A1 consensus sequence with 646T>A encoding Phe216Ile [212,261,293]. Twenty-one Ax alleles are listed in dbRBC; six of them (Ax01−Ax06) encode Phe216Ile [215].

Red cells from group A babies usually react only weakly with Dolichos lectin and may not be agglutinated at all by human anti-A1. 9 A1A2BO genotypes and serologically determined phenotypes. Genotype Phenotype A1/A1 A1/A2 A1/O A1 A2/A2 A2/O A2 B/B B/O B A1/B A1B A2/B A2B O/O O strong Sda antigen and Tn polyagglutinable red cells, regardless of ABO group (Chapters 31 and 33). A2 red cells have substantially higher expression of H antigen than A1 cells. 9). 1 A1- and A2-transferases (GTA1 and GTA2) and the genes that produce them A-transferase (GTA) isolated from sera or gastric mucosa of A1 individuals is more effective at converting group O red cells to A-active cells than that from A2 people [205– 208].

Sequencing of the ABO genes showed that the child had an ABO gene in which exon 6 (and, presumably exons 1–5) had the sequence of a B allele and exon 7 the sequence of an O1 allele. This hybrid gene had probably arisen in the germline of the mother as a result of crossing-over during meiosis. This B-O1 gene would encode an enzyme with GTA activity because O1 and A1 have an identical sequence in exon 7, the region encoding the catalytic site; the absence of 261delG in exon 6 of B origin enables translation of this active enzyme.

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