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Additional info for Omega-3 Fatty Acids. Chemistry, Nutrition, and Health Effects

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The reason that LDL-C does not typically decrease with n-3 HUFA consumption would appear to be related to the measured depression in LDL receptor activity induced by these fatty acids when consumed in atypical amounts, eg. 10 g per day (21,22). Thus, the decrease in LDL formation, coupled with a decrease in LDL clearance, combine to leave LDL-C relatively unchanged, or even slightly elevated. ch004 g Figure 12. Exchanging 2% energy from 18:2n6 with n3 HUFAs (EPA & DHA) and lack of effect on TC or the LDL/HDL ratio in humans (from ref.

The result produced no change in TC, but the LDL/HDL ratio improved because LDL slightly decreased as HDL rose 7%, not unlike the human results of Sundram et al. (1 7). These empirical results suggest that the "metabolic tension" between SFAs and PUFAs is an important determinant of the LDL/HDL ratio. Furthermore, in addition to the total fat intake, the absolute intakes of 18:2 and SFAs have a critical bearing on the total plasma cholesterol, such that reducing only SFAs increases the P/S ratio of the diet and decreases TC, LDL-C as well as HDL-C if the fatty acid balance becomes overly distorted.

Pronczuk, A. Hayes, K. C. J. Nutr. 1998, 128, 477-84. 5. ; Pronczuk, A. C. J. Nutr. Biochem. 1995, 6, 353361. 6. Hayes, K. C. Khosla, P. FASEB J 1992, 6, 2600-2607. 7. ; Hayes, K. C. FASEB J 1994, 8, 1191-1200. 8. Hayes, K. ; Khosla, P. J. Nutr. Biochem. 1995, 6, 188-194. 9. C. Can. J. Cardiol. 1995, 11, 39G-46G. 10. Baudet, M . ; Lasserre, M . ; Esteva, Ο. Jacotot, Β. J. Lipid Res. 1984, 25, 456-468. 11. ; Grundy, S. M . J. Lipid Res. 1985, 26, 194-202. 12. ; Merinero, M . ; Sanchez-Muniz, F.

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