By Fix J.D.

Now in its moment variation, this e-book extracts an important info on neuroanatomy and offers it in a concise, uncluttered type to organize scholars for the USMLE. High-YieldT capacity precisely that!

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T h e hypothalamospinal tract at T-1and above. Damage results in ipsilateral . , miosis, ptosis, herniinhidrosis, and apparent e n o p h thalmos). 5. Tile ventral (anterior) horn. Damage results in ipsilateral flaccid paralysis of innen-ated muscles. B. Ventral spinal artery occlusion (see Figure 8-2F) causes infarction of h e anterior c \ x 7 0 thirds of the spinal cord, but spares the dorsal columns and horns. T h e lateral corticospinal tracts. Damage results in bilateral spascic paresis with ~)'ramidalsigns beloti.

T h e spinocerebcllnr tracts. Darnage rcsults in bilateral arm nnd leg dystasia. D. Syringornyelia (see Figure 8-2H) is n central cavitation of the cenlical cord o f u n k n o w n etiology. I t rcsults in darnagc to the following structures: 48 Chapter 8 1;The v e n m l ~ 1 1 i tcomn~issure. e D ; ~ m n gto J e c u s r ~ t i n glateral spinothalamic ;I> or~s'cnirsesl~il;~rer;~l loss of pain and terliperatitre sensation. 2. LAIN lesions result in flaccid paralysis of the intrinsic tni~sclc ofr h c 11nncls.

A r n a ~results e in bilateral spastic paresis with pyramidal signs. 3. T h e spinocerebcllnr tracts. Darnage rcsults in bilateral arm nnd leg dystasia. D. Syringornyelia (see Figure 8-2H) is n central cavitation of the cenlical cord o f u n k n o w n etiology. I t rcsults in darnagc to the following structures: 48 Chapter 8 1;The v e n m l ~ 1 1 i tcomn~issure. e D ; ~ m n gto J e c u s r ~ t i n glateral spinothalamic ;I> or~s'cnirsesl~il;~rer;~l loss of pain and terliperatitre sensation.

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