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Compiled results demonstrate that 82% of quinagolide-treated patients achieved normal PRL levels, compared with 71% of those who received BC. Only 7% stopped quinagolide for side effects, compared with 23% with BC (Webster, 1996). A randomized controlled crossover trial examined 20 patients treated with either CAB or quinagolide, followed by a washout period with placebo and subsequent treatment with the other of the two drugs (Di Sarno, et al; 2000). Eight of the patients had microadenomas, six empty sella syndromes and six idiopathic HP.

Karavitaki N, Thanabalasingham G, Shore HC, Trifanescu R, Ansorge O, Meston N, Turner HE, Wass JA. (2006) Do the limits of serum prolactin in disconnection hyperprolactinaemia need re-definition? A study of 226 patients with histologically verified non-functioning pituitary macroadenoma, Clin Endocrinol (Oxf) 65, pp. 524–529. Katz E, Schran HF, Adashi EY. (1989) Successful treatment of a prolactin producing macroadenoma with intravaginal bromocriptine mesylate: a novel approach to intolerance of oral therapy.

32, pp. 177–182. Bole-Feysot C, Goffin V, Edery M, Binart N, Kelly PA. (1998) Prolactin (PRL) and its receptor: actions, signal transduction pathways and phenotypes observed in PRL receptor knockout mice, Endocr Rev 19:225. Bracero N and Zacur HA. (2001) Polycystic ovary syndrome and hyperprolactinemia, Obstet Gynecol Clin North Am 28, pp. 77–84. (1989) Cerebrospinal fluid rhinorrhea occurring in long-term bromocriptine treatment for macroProlactinomas. Surg Neurol. 32(5):346-9. Hyperprolactinemia and Woman’s Health 35 Brooks DJ.

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